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For Physicians, Psychiatrists, and Neurobiological ResearchersEMDR has been given the highest effectiveness rating for the treatment of trauma in the recent American Psychiatric Association Practice Guideline for the Treatment of Patients with Acute Stress Disorder and Posttraumatic Stress Disorder. Arlington, VA: American Psychiatric Association Practice Guidelines, 2004. As noted on the Home Page, health departments of numerous countries have designated EMDR as a first line treatment for PTSD: http://www.EMDRHAP.org/researchandresources.htm EMDR's theory and practice attends to somatic, as well as emotional and cognitive factors of the clinical condition. EMDR is an integrative form of psychotherapy, which includes aspects of, but is distinct from, psychodynamic and cognitive behavioral therapies. For instance, Albert Ray, MD, past president of the American Academy of Pain Medicine, indicates that EMDR benefits patients with chronic pain in ways different from, and potentially surpassing those, found in current CBT treatment. Ray, A. L. & Zbik, A. (2001). Cognitive behavioral therapies and beyond. In C. D. Tollison, J. R. Satterhwaite, & J. W. Tollison (Eds.) Practical Pain Management (3rd ed.; pp. 189-208). Philadelphia: Lippincott. An information processing model is used to explain EMDR's clinical effects. This model is not linked to any specific neurobiological mechanism since the field of neurobiology is as yet unable to determine the neurobiological concomitants of any form of psychotherapy, nor many medications. However, since EMDR achieves clinical effects without the need for homework, or the prolonged focus used in exposure therapies, attention has been paid to the possible neurobiological processes that might be evoked. Harvard researcher, Robert Stickgold, MD, has explained how EMDR may directly tap into the processes of Rapid Eye Movement (REM) sleep: Stickgold, R. (2002). EMDR: A putative neurobiological mechanism of action. Journal of Clinical Psychology, 58, 61-75. Numerous investigations have examined the neurobiological aspects of the eye movement component. Andrade, J., Kavanagh, D., & Baddeley, A. (1997). Eye-movements and visual imagery: a working memory approach to the treatment of post-traumatic stress disorder. British Journal of Clinical Psychology, 36, 209-223.Tested the working memory theory. Eye movements were superior to control conditions in reducing image vividness and emotionality. Barrowcliff, Gray, Freeman, MacCulloch (in press) Eye-movements reduce the vividness, emotional valence and electrodermal arousal associated with negative autobiographical memories. Journal of Forensic Psychiatry and Psychology.Tested the reassurance reflex model. Eye movements were superior to control conditions in reducing image vividness and emotionality. Barrowcliff, A.L., Gray, N.S., MacCulloch, S., Freeman, T. C.A., & MacCulloch, M.J. (2003). Horizontal rhythmical eye-movements consistently diminish the arousal provoked by auditory stimuli. British Journal of Clinical Psychology, 42, 289-302.Tested the reassurance reflex model. Eye movements were superior to control conditions in reducing arousal provoked by auditory stimuli. Christman, S. D., Garvey, K. J., Propper, R. E., & Phaneuf, K. A. (2003). Bilateral eye movements enhance the retrieval of episodic memories. Neuropsychology, 17, 221-229.Tested cortical activation theories. Results provide indirect support for the orienting response/REM theories suggested by Stickgold (2002). Saccadic eye movements, but not tracking eye movements were superior to control conditions in episodic retrieval. Kavanagh, D. J., Freese, S., Andrade, J., & May, J. (2001). Effects of visuospatial tasks on desensitization to emotive memories. British Journal of Clinical Psychology, 40, 267-280.Tested the working memory theory. Eye movements were superior to control conditions in reducing within-session image vividness and emotionality. There was no difference one-week post. Kuiken, D., Bears, M., Miall, D., & Smith, L. (2001-2002). Eye movement desensitization reprocessing facilitates attentional orienting. Imagination, Cognition and Personality, 21, (1), 3-20.Tested the orienting response theory related to REM-type mechanisms. Indicated that the eye movement condition was correlated with increased attentional flexibility. Eye movements were superior to control conditions. Sharpley, C. F. Montgomery, I. M., & Scalzo, L. A. (1996). Comparative efficacy of EMDR and alternative procedures in reducing the vividness of mental images. Scandinavian Journal of Behaviour Therapy, 25, 37-42.Results suggest support for the working memory theory. Eye movements were superior to control conditions in reducing image vividness. Van den Hout, M., Muris, P., Salemink, E., & Kindt, M. (2001). Autobiographical memories become less vivid and emotional after eye movements. British Journal of Clinical Psychology, 40, 121-130.Tested their theory that eye movements change the somatic perceptions accompanying retrieval, leading to decreased affect, and therefore decreasing vividness. Eye movements were superior to control conditions in reducing image vividness. Unlike control conditions, eye movements also decreased emotionality. Additional Neurobiological Indicators Lamprecht F, Kohnke C, Lempa W, Sack M, Matzke M, Munte T. (2004). Event-related potentials and EMDR Treatment of post-traumatic stress disorder. Neuroscience Research, 49, 267-272. Lansing, K., Amen, D.G., Hanks, C. & Rudy, L. (in press). High resolution brain SPECT imaging and EMDR in police officers with PTSD. Journal of Neuropsychiatry and Clinical Neurosciences. Levin, P., Lazrove, S., & van der Kolk, B. A. (1999). What psychological testing and neuroimaging tell us about the treatment of posttraumatic stress disorder (PTSD) by eye movement desensitization and reprocessing (EMDR). Journal of Anxiety Disorders, 13, 159-172. van der Kolk, B., Burbridge, J. and Suzuki, J. (1997). The psychobiology of traumatic memory: Clinical implications of neuroimaging studies. Annals of the New York Academy of Sciences, 821, 99-113. |
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